Ignacio Palmero graduated in Biological Sciences at the Universidad Complutense de Madrid and obtained a PhD degree in Molecular Biology from the Universidad Autónoma de Madrid.
He did postdoctoral stays at the Imperial Cancer Research Fund, London, UK and Centro Nacional de Biotecnología, Madrid, Spain. In 2002 he obtained a staff scientist position of the Spanish Research Council (CSIC) at the Institute for Biomedical Research Sols-Morreale, Madrid. The main focus of his research is to understand the program of cellular senescence and how its malfunction leads to human disease, with the aim to exploit this knowledge for diagnosis and therapy.
Throughout his career, Ignacio Palmero has made significant contributions to this field, including the identification of the ARF-p53 axis in oncogene-induced senescence, the role of senescence in fibroblast plasticity and, more recently, the link between embryo senescence and developmental disease. He is a member of the International Cell Senescence Society, ICSA, and has participated in the Scientific Committee of several ICSA meetings. He currently serves as member of the Steering Committee of the Spanish Society of Cell Senescence, Senescel.
Nuria Casa Rodríguez completed her bachelor’s degree in Biology in 2018 at Complutense University of Madrid. In 2019, she did a Master’s degree in Genetics and Cell Biology at the Complutense University of Madrid, Autonoma University of Madrid and the University of Alcalá. In 2021 she completed an MSc in “Cancer Research & Precision Oncology” at Glasgow University.
During these last few years, she has done different research placements in Spain and abroad. She worked in the Center for Biological Research (CIB, CSIC), and the Spanish National Cancer Research Center (CNIO) in Madrid, Spain and at the CRUK Beatson Institute, Glasgow, UK.
Nuria joined the Cellular Senescence and Tumor Suppression lab at the Instituto de Investigaciones Biomédicas Sols-Morreale (IIBM, CSIC-UAM) in 2022, where she is currently conducting her PhD research. Her work focuses on the study of how the dysfunction of the physiological program of cellular senescence during embryogenesis can lead to developmental defects.